CAS NO.: 120287-85-6
Molecular formula:C70H92ClN17O14
Synonyms:Cetrorelix;Cetrorelixum
Shelf life: 24 months
Purity : 98% HPLC
Certificate:COA; HPLC; MSDS; TDS,etc.
MOQ : 500g,1kg
Shipping: DHL/FedEx/UPS/USPS Safe and fast delivery
Lead Time: 1-3 days
Warehouse: USA and Germany warehouse" data-aw-url="https://www.faithfulbiochem.com/steroid-raw-powder/cetrorelix-acetate-peptide-powder/" title="Cetrorelix acetate peptide powder">
Xi'an Faithful BioTech Co., Ltd. is a leading peptide supplier in northwest China, Our Cetrorelix acetate peptide powder adopts advanced production processes and purification technologies to accurately control key factors such as production temperature and humidity. Ensure that all aspects of the product comply with international standards. Provide high-quality raw materials for global customers, from early detection, preclinical research and clinical development to commercial production.
Cetrorelix acetate peptide powder is a synthetic peptide mainly used in assisted reproductive technology to prevent premature ovulation in patients undergoing controlled ovarian stimulation. Cetorelix acetate is a gonadotropin-releasing hormone (GnRH) antagonist that can competitively bind to receptors on the pituitary cell membrane with endogenous LHRH (CAS 9034-40-6), thereby inhibiting pituitary secretion of luteinizing hormone (LH) and follicle stimulating hormone (FSH) in a dose-dependent manner. Xi'an Faithful BioTech Co., Ltd's Cetrorelix acetate has high purity and minimal impurities, which can meet your research or production needs.
Test | Specification | Result |
Appearance | White or off-white powder | White powder |
Amino acid analysis | 2-Nal:0.9-1.1 | 1 |
4-Cpa:0.9-1.1 | 1 | |
3-Pal: 0.9-1.1 | 1 | |
Ala:0.9-1.1 | 1.1 | |
Tyr:0.9-1.1 | 0.9 | |
Leu:0.9-1.1 | 1.1 | |
Arg:0.9-1.1 | 1 | |
Pro:0.9-1.1 | 1 | |
Ser:Present | Present | |
MS | 1431.0±1.0 | 1431.1 |
Related Substances | Unspecified impurity for each impurity: NMT1.0% | 0.14%(single largest impurity) |
Total impurity: NMT 2.0% | 0.41% | |
Acetic acid | NMT 15.0% | 6.80% |
Water Content(Karl Fischer) | NMT 7.0% | 4.80% |
Peptide Content(N%) | NLT80% | 88.10% |
Bacterial endotoxins | Less than 5IU/mg | Conforms |
Residual solvent | Methanol:NMT 3000ppm | Conforms |
Acetonitrile:NMT 410 ppm | ||
Triethylamine:NMT 320 ppm | ||
Conclusion:Conform with enterprise specification |
Always prioritize product quality and make every effort to ensure high purity and stability of the products.
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In treatments such as in vitro fertilization (IVF) or intracytoplasmic sperm injection (ICSI), it is used to inhibit the premature appearance of endogenous luteinizing hormone (LH) peaks, prevent premature ovulation of follicles, prolong follicle development time, and improve the success rate of egg retrieval.
By antagonizing GnRH receptors, inhibiting excessive LH secretion, improving ovulation disorders caused by hyperandrogenism in PCOS patients, and assisting with the use of ovulation inducing drugs.
In the ART cycle, by inhibiting endogenous LH peaks, the incidence of OHSS caused by exogenous gonadotropin stimulation is reduced, especially for high-risk populations such as polycystic ovary syndrome or high response patients.
Cetrorelix acetate may be used in artificial insemination (IUI) or other reproductive technologies, and medication needs to be adjusted according to individualized plans.
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Q1:What are the common side effects of Cetrorelix acetate?
A1:Side Effects incuding Abdominal or stomach pain,continuing or severe nausea, vomiting or diarrhea,decreased amount of urine,feeling of indigestion,moderate to severe bloating,pelvic pain, severe,rapid weight gain,shortness of breath.
Q2:What payment method do you support?
A2: we support Bank Transfer.Wechat and Alipay.
Q3:How it works?
A3: It is the competition of endogenous GnRH for receptors on pituitary cells, thereby inhibiting the release of endogenous luteinizing hormone (LH) and follicle stimulating hormone (FSH), delaying the appearance of LH peak, and controlling ovulation. Its function is dose-dependent, and its inhibitory effect is direct and maintained through continuous treatment, without causing an initial increase and subsequent decrease in progesterone receptor activity.
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